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Durabolin was first known under the name nandrolone phenylpropionate. Despite the lack of any known therapeutic applications, the drug remained legal until 2001. Finally, we recommend considering potential side effects of AAS in further in vivo studies: changes in cholesterol levels (increased low-density lipoprotein and decreased high-density lipoprotein), liver damage, nephropathy, cardiovascular pathologies as well as conditions pertaining to hormonal imbalance have been reported in response to AAS high-dose or prolonged administration.

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Insulin (secondary to carb consumption) inhibits protein breakdown with no impact on protein synthesis. Previous studies have shown that acute exposure to the AAS for non-neuronal cell lines in vitro inhibits aromatase activity in a dose-dependent manner (Mor. This suggested hydroxylation of steroidal skeleton and oxidative cleavage of heptanoate moiety.

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In contrast to the case of testosterone, such potentiation occurs to a reduced extent or not at all with most synthetic AAS (as well as with DHT), and this is primarily responsible for the dissociation of anabolic and androgenic effects with these agents.

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